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How long does shipment process take?
Estimated deliverance time for our shipment is max. 5-10 business days within USA, and 7-21 business days to Europe and the rest of the world. However, this shipping time will depend on various factors such as postal service efficiency, customs clearance, international transit, etc., which is why we give you an approximation based on our statistics and former experience.

How are products shipped?
We ship in discreet..... EDITED. We are sending only recorded/certified packages (that mean that packages passed the custom control) and it is can not happened to not get your package. We ensure that all packages are shipped with maximum discretion. We wrap items in .... EDITED. Xray will not penetrate. Our organization name is not indicated on any package and shipments are made from various shipping points in USA and Europe to ensure maximum discretion and highest probability of successful delivery.
With customs we have 99.9% success to USA, 99.5% success to Canada and Europe and 97% to Australia. Even if products get seized ( 1% possibility based on our statistics through years), we cover 100% of the loss.

How are bulk/large quantity orders shipped?
If the order is large enough to not fit in the same shipment envelope as a small sized order, we guarantee the order will be divided into the necessary amount of packages. No matter how large your order is, we reassure you the same discretion level and repackaging is used as with a small sized order.

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A prescription is required  to comply with local law.

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Minimal order is ZERO $0 (NULL), a maximum - no limits.

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We have two methods of shipping - by regular mail and express. Shipping costs $10 for regular and $200 for express no matter how big your order is.

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Yes, we ship to P.O. Boxes.

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We ship worldwide.

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Payments made by Western Union/ Money Gram are normally processed and cleared within 1-3 business day. Confirmation of payment will follow once payment has cleared. Cash in the mail and greendot can take a little more.

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Since laws differ from country to country, we recommend you to contact your local FDA or customs office regarding the regulations and restrictions of your country.

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We are a professional mail order service licensed to sell and distribute pharmaceutical products. We have been established for over 8 years, supplying original, high-quality anabolic steroids and bodybuilding related products, prescription-free over the Internet.

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Since we are specialized in internet mail ordering, our services are only available through our web site.

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All products have been tested in laboratories in United States of America, Canada & Europe. Everything is 100% legit - the pictures on our web site are the exact products you are going to get.

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We don`t send them anymore since we have had losses, you can be sure that you are ordering No.1 quality products.

What about discounts for bigger orders?
We do offer discounts for bulk orders. We are always willing to make a better deal for your order if it is bigger.

T3 and the Modern Athlete

So you've decided to use T3 to help you shed fat now that you've read up on it and gotten past the nay Sayers who expound the ills of shutting down your body’s own production of natural thyroid. Wonderful, T3 when used correctly can be a great addition to any diet and cardio plan. Read that again boys and girls, IN ADDITION TO ANY DIET AND CARDIO PLAN!!! If you've turned to T3 because you think it's a magic pill that will allow you to eat like crap and still lose weight you've been listening to the wrong advice. Can you lose weight/fat while using T3 and still eating junk food, unfortunately yes to a degree. I say unfortunately because this fact often leads people to do just that, it starts with a cheat meal that turns to a cheat day, which eventually has the athlete eating whatever and whenever they want and still they lose some weight. So what's wrong with this if the eventual out come, weight loss that is, is reached? The first problem is the weight you are losing may not be fat if your eating like crap, the second is what happens when you stop the T3 cycle and your metabolism is suppressed temporarily, if you were eating sloppy during the T3 usage your most likely to keep following that pattern and the combination of a slow metabolism combined with sloppy eating results in rebound weight gain. So in the end what have you really accomplished outside of being able to eat what you want with out getting any fatter for a month or so? And that's if you’re lucky and the rebound weight gain doesn't push you past your starting weight!!!

Now that I have your attention and you know what NOT to do, let's concentrate on what TO do. Just like any other chemical we find in our arsenal, T3 can and is used in a variety of ways when it comes to dosage and length of cycle, both for cutting and bulking. This article will deal with cutting use only. There are some who prefer to "hit it hard" and go high dosage with a quick taper down at the end losing a great amount of weight in a short time, but this way tends to eat as much muscle as fat in my experience and you end up looking basically the same as when you began, except that you weigh less and are smaller. There are those who like to use the same dosage throughout the cycle with no taper up or down figuring if your metabolism is going to be sluggish anyhow why waste the days using it at a low dosage when you could be burning more fat on those days. Then there are those who slowly taper up, maintain the highest dosage for a set time and then slowly taper down. It's the last group I'll concentrate on here, as this is the system that has shown it's best overall results with those I've worked with.

Let's start with the dosage, T3 is a very individual drug, when it comes to dosage I've seen guys use as high as 250-300mcg/day and others as low as 25mcg/day where both athletes lost fat and reached their goals. As a rule I start everyone (and for now I'm dealing with men I'll pen an article on women’s usage in the future) at 25mcg/day. I usually base the time of the cycle on their individual weight loss goals, if it's a smaller amount I'll go 3 weeks tops, if it's a lot of weight to lose we'll go 4, 5 and sometimes 6 weeks. I generally don’t go over 6 weeks with anyone, as T3 tends to stop working in most people after that amount of time. I’d rather they run 4 weeks cycles with 2 weeks off where they use an ECA stack or Clen during the break to continue to lose fat, then run another 4 week cycle. So the 1st 3 days in this cycle would be 25mcg/day, then the 2nd 3-day period is 50mcg/day, etc. The typical 21-day cycle will look like this:

	Day 1	Day 2	Day 3	Day 4	Day 5	Day 6	Day 7
Week 1	25 mcg	25 mcg	25 mcg	50 mcg	50 mcg	50 mcg	75 mcg
Week 2	75 mcg	75 mcg	100 mcg	100 mcg	50 mcg	75 mcg	75 mcg
Week 3	75 mcg	50 mcg	50 mcg	50 mcg	25 mcg	25 mcg	25 mcg

As you can see the dosage is increased by 25mcg/day every 4th day until the maximum dosage is reached for the subject, in this case 100mcg/day, then lowered the by the same 25mcg/day increments every 4th day until the end of the cycle. Given that most of the people I've worked with have tried everything else and are still considerably overweight when they start, the full 4-week cycle is often used instead of the 21-day cycle. The one I've used lately with the most success is as follow, remember the jumps are still 25mcg/day but this time you increase/decrease the dosage every 4 days:

	Day 1	Day 2	Day 3	Day 4	Day 5	Day 6	Day 7
Week 1	25 mcg	25 mcg	25 mcg	25 mcg	50 mcg	50 mcg	50 mcg
Week 2	50 mcg	75 mcg	75 mcg	75 mcg	75 mcg	100 mcg	100 mcg
Week 3	100 mcg	100 mcg	75 mcg	75 mcg	75 mcg	75 mcg	50 mcg
Week 4	50 mcg	50 mcg	50 mcg	25 mcg	25 mcg	25 mcg	25 mcg

Note: You could also do the 3-day increase/decrease and hold the maximum dosage of 100mcg/day for days 10-19, but some find 100mcg/day makes them too uncomfortably warm and they sweat too much, especially during the warmer months.

There you have it, simple yet effective. If you remember to supplement your diet with plenty of protein (which every lifter should anyhow), eat a clean calorie controlled diet, drink 1-2 gallons of water per day and to take a mild steroid cycle to minimize muscle loss you should be able to see rapid fat loss with this cycle. I should also mention that some people like to stack T3 with Clenbuterol for even better results. I’ve purposely left this out as I will be including Clen in Part 2 of this series. Good luck and may you all reach your cutting goals!!!

Thyroid Suppression
Let’s start with the biggest misconception still around where T3 is concerned, that is suppression of natural thyroid output. I’m amazed that this drug has been used now for the past several years by literally thousands of athletes with few if any reported cases of thyroid shutdown yet the 1st thing someone says when a person asks about T3 is “it will shut down your natural thyroid and you’ll be on T3 the rest of your life”. Numerous studies have been done and show that cessation of exogenous T3 does not shut down your natural thyroid. The 1st study was done in 1951 by M. Greer (1) and showed that patients that were misdiagnosed as hypothyroidism that later had their medicine withdrawn showed no shutdown of their natural thyroid as their thyroid returned to normal within 2 weeks. His studies also showed that it didn’t matter if the patients thyroid had been medically suppressed for 30 years or a few days they both returned to normal within two weeks. Hence my mentioning in Part 1 of a sluggish thyroid post T3 cycle and my suggestion that you continue to eat clean, do cardio and use a fat burner like the Ephedrine/Caffeine/Aspirin stack, Clenbuterol or an over the counter fat burner until your natural thyroid output returns to normal. Numerous studies have been done since Greer’s that have confirmed his findings. As with any medicine there are always exceptions to the rule and there have been a few people who claim to have had their own thyroid function permanently damaged by T3 usage but in my experience this only occurred when ridiculously high dosages were used, if you adhere to the dosages recommended here in you should be fine.

T3/Clenbuterol Cycle
This has to be the most often used cutting combo used today for fat loss in weight trained athletes, or at least the most talked about. Both drugs when used on their own are effective fat burners through differing pathways, but used together they have a synergistic effect and create a very potent fat burning cycle. The medical reasoning for this is long and complicated and not necessary to understand at this point but it is out there for anyone to research should you need to know, in simple terms each not only do their own job but also help the other’s fat burning process so that in effect, as they say, 1+1=3. So what dose do you use for each drug? For the T3 I suggest you use the same dose scheme I outlined in Part 1, again I took some flack over the lower dosing as some feel you should go higher but as I said from my experience anything over 75mcg-100mcg/day (for men, women’s dosage should go no higher than 50mcg/day) usually burns much too much muscle tissue in addition to fat tissue, unless that is your goal I would stay with as small a dose as you can get away with where you can still tolerate the increased body temp, for most men that is 75-100mcg/day, for most women that is 50mcg/day max. *Using Clen will increase your body temp also so you will have to monitor both drug dosages to see what you can comfortably tolerate. Clenbuterol dosing is a very individual thing, some cycles recommend 160mcg/day at the maximum dosage some 80mcg/day but the 1 thing most agree on is to start low and ease your dosage upwards as you feel comfortable with it the 1st time you use it. With subsequent cycles you can start at your maximum tolerable dose or slightly lower and then increase the dosage over a few days until you reach your maximum again as some people report the maximum they can use differs from 1 cycle to another. Which brand and whether you use tabs vs. liquids could also have something to do with the differing max doses. I would suggest you start your 1st cycle of Clen with 20mcg/day and increase by 20mcg/day until you reach the upper maximum you can use based on the side effects. The most common side effects are shaking, jitteriness, anxiety and raising of body temperature, basically the feelings you get when you’ve had way too much caffeine or cold medication are what your looking out for. When those sides get to be too much cut back to the last tolerable dose. A popular Clenbuterol cycle is 2 weeks on, 2 weeks off. For men I would suggest starting at 20mcg/day and going up to 100-120mcg/day or like I said whatever you can tolerate, stay there until day 14 then end the cycle, women should try half that max dose but if you can tolerate more and want to use it then go for it this is definitely a trial and error process. Take 2 weeks off and then repeat if desired, again starting at or near your maximum dose that you figured out with the 1st cycle. When stacking with T3 the question becomes what do you do on the 2 weeks your off Clen but still are on T3? That’s really an individual decision for you to make, you could rotate an ECA stack or a Gugglesterone with the Clen cycle so that your doing 1 for 2 weeks then the other for 2 weeks. Or you could simply take 2 weeks off after the end of the Clen where your only on the T3 for the next 2 weeks, you’ll be at your mid to max dosage of T3 by then so you’ll still be burning fat just fine. Then after the 4 weeks of T3 you’ll be done with both the T3 and the Clen and you could start a ECA stack for 2 weeks if you are ending the cutting cycle and want to protect yourself against rebound weight gain while waiting for your natural thyroid levels to return to normal. If you have more fat to lose you can cycle off T3 for 2 weeks as I said in Part 1 and repeat the cycle again. When to use Clen again will depend on when you used it last, remember 2 weeks on, 2 weeks off. There’s nothing to say you can’t cycle T3 ad ECA together while you wait to add the Clen back in, just remember whenever you come off the T3 you want something in your system to help burn fat while you wait for your natural thyroid to return to normal. Also remember that Clen cycles are like T3 cycles in that there’s several different cycle’s currently popular, and you’ll most likely get different advice to the length and type of cycle by asking more than one person. The advice I give is based on those I’ve had use it and report back to me their results and feeling on it. I’m all for experimenting but until something comes along that proves to be better I’ll stick with the 2 weeks on/2 weeks off cycle advice where Clen is concerned.

T3 in Bulking Cycles
I briefly touched on using T3 in bulking cycles and many members seemed confused as to how a fat burner could help with a bulking cycle. T3 is a drug mainly known for raising one’s metabolism and burning fat, and possibly muscle tissue, when used at higher dosages (> 75-100 mcg/day for men, > 50mcg/day for women), but at lower dosages (12.5-25mcg/day for men, ½ that for women) it causes a faster conversion of carbohydrates, proteins, and fats. It’s the increased conversion and absorption of nutrients that increases the results of your bulking cycle when you use it with a bulking cycle. When you run a bulking cycle you do so in conjunction with a higher protein/higher calorie diet because we know in order to grow muscle we need to feed the body nutrients, so there are plenty of nutrients to be converted, thus the bulking cycle gets a “push” if you will yielding better results. I can tell that literally every single person who has taken my advice and tried using a small amount of T3 daily with their bulking cycle has reported better gains than they usually get without it. I’ve even had success using 25mcg/day every other day with a bulking cycle. When you consider the low cost of T3 at such small a dosage it’s definitely a cheap insurance to better gains.

Women’s Cycles
Although women have been known to use T3 with good success I always hesitate to recommend a cycle to them for the simple reason that women seem to be much more sensitive to T3 than men are. The rebound weight gain can be significant if the post T3 period isn’t monitored stringently and an over the counter fat burner isn’t used. That said if you’re still set on using it here is a simple straightforward 21 day cycle, again using the 3 day ramp up and ramp down method.

	Day 1	Day 2	Day 3	Day 4	Day 5	Day 6	Day 7
Week 1	12.5 mcg	12.5 mcg	12.5 mcg	25 mcg	25 mcg	25 mcg	37.5 mcg
Week 2	37.5 mcg	37.5 mcg	50 mcg	50 mcg	50 mcg	37.5 mcg	37.5 mcg
Week 3	37.5 mcg	25 mcg	25 mcg	25 mcg	12.5 mcg	12.5 mcg	12.5 mcg

If you want to run it longer than 21 days, you can add in more days at the maximum dosage or use it in 4 day blocks with the ramp up and ramp down. Again please remember women are more sensitive to T3 than men and the rebound weight gain can be much more significant if your not ultra vigilant with the post T3 period, keep eating a very clean diet with calories below maintenance, and use either Gugglesterones, ECA stack or any other over the counter fat burner you feel comfortable with to help boost your natural metabolism until your system recovers, which could be anywhere from a few days to about 2-3 weeks.

Dosage Timing
T3 has a ½ life that doesn’t necessitate multiple daily dosing, so taking your entire daily dose at once is usually recommended. That said if your cycle requires you to take 100mcg/day or more I usually recommend splitting the dosage in ½ and taking it twice per day just to insure if you are sensitive to the drugs possible side effects you limit the exposure. Again I would suggest taking it in the morning, then around dinner time if a 2nd dose is necessary. I know that for myself, certain brands cause an upset stomach if I take more than 50mcg at a time, so at 75-100mcg/day I’d split it into 50mcg in the morning and the balance at nighttime.

Rebound Weight Gain
Rebound weight gain is inevitable when using T3, the best you can hope for is to minimize it. A good start is to make sure you use at least a small amount of a steroid with the cycle, this will help you to hold on to the muscle mass you already have. The best thing you can do is to take a post cycle over the counter fat loss product such as ECA stack, Gugglesterones or some other similar product. What your looking for here is the continuance of the fat loss while your system returns to your normal thyroid output. This should occur with in 2-3 weeks, so during that time continue to eat clean, do cardio, drink plenty of water and take the over the counter fat loss product. You’ll know when your thyroid has returned to normal when your body temp returns to normal. Women are especially warned to be very vigilant here, most people are eager to eat more when their cycle ends but this is not the time when using T3, you need to make sure your metabolism has been restored before splurging a bit.

 

Omnadren 250 (Testosterone mix)

Generic name:

Testosterone Propionate
Testosterone Phenylpropionicum
Testosterone Isocapronicum
Testosterone Caprinicum


Poland, Jelfa SA
(pharmacy production)
pack: 5 vials a 1ml 250mg/ml

Active Life: Approx. 18 days
Drug Class: Anabolic/Androgenic Steroid (for injection)
Average Dose: 250-1000 mg/week (males only)
Acne: Yes
Water Retention: Yes
High Blood Pressure: Yes
Liver Toxic: Low
Aromatization: Yes
DHT Conversion: Yes, high
Decrease HPTA function: Yes, severe

Omnadren 250 is an oil-based injectable containing a blend of four different testosterone esters: Testosterone propionate, phenylpropionate, isocaproate and caproate. Being as it is a four-ester testosterone, Omnadren is most commonly compared to Sustanon. While it does contain Testosterone propionate, testosterone phenylpropionate and isocaproate in the same strength as Sustanon, the last ester is different. Please note however, that the older versions of Omnadren list isohexanoate and hexanoate as the final two ingredients. Hexanoate is simply another word for caproate, so the last ester (decanoate) is the only Sustanon constituent missing from Omnadren.

One of the only noticeable differences between Sustanon and Omnadren seems to be the speed in which estrogen buildup occurs. In comparison, the process appears to be slightly more pronounced with Omnadren. This is of course just a matter of timing, as the slowest releasing ester in Omnadren (caproate) is a little faster acting than enanthate. Blood testosterone levels will therefore peak much faster with this compound, not having the same gradual release time imposed by testosterone decanoate. Users likewise report water retention much earlier into a cycle. While water retention may lead to a more rapid buildup of size and strength, it can become pronounced enough to cause a very smooth and watery look to develop (hiding muscle definition). In addition, the excess estrogen is likely to cause the development of gynecomastia. This effect is especially pronounced with Omnadren, usually presenting itself quickly after a cycle has been started. Estrogen can also be responsible for increases in body fat storage during treatment, resulting in a further loss of definition. Individuals who are sensitive to the effects of estrogen, yet still seek the power of a testosterone, would therefore need to add an anti-estrogen such as Nolvadex and/or Proviron. Arimidex,Femara, or Aromasin, 3 powerful anti-aromatase, are another option available to us. These three drug works much more efficiently than any other anti-estrogen. They would have great use with such a strong item as Omnadren, as the standard remedies would not be quite as effective.

Being a testosterone, you can also expect the typical set of androgenic side effects. Oily skin, acne, body/facial hair growth and increased aggression are all very common with this product. It can also bring out or aggravate a condition of male pattern baldness. We do however, have the option to addition Proscar (finasteride). This is a drug that can effectively prevent testosterone from converting into DHT (dihydrotestosterone) in certain androgen target tissues. Since DHT is the primary culprit with testosterone's androgenic side effects, adding Proscar to the cycle should allow it to be much more comfortable. Omnadren is also likely to suppress endogenous testosterone production rather quickly. It is therefore a necessity to add a testosterone stimulating drug like HCG and/or Clomid/Nolvadex when concluding therapy.

Being a powerful, long acting testosterone blend, the effect of Omnadren is of course quite comparable to that of Sustanon (except that its release time is closer to cypionate or enanthate). It is similarly a powerful androgen, capable of providing great gains in mass and strength. Due of the high level of water retention associated with testosterone, Omnadren is really only applicable for bulking purposes. While it is very effective alone, it is also combined often with a number of other steroids depending on the desired result. Many athletes prefer to combine Omnadren with a strong anabolic like Deca-Durabolin or Equipoise for example, in an attempt to lower the overall testosterone dosage and run a more quality mass building cycle. On the other hand, power-lifters and those looking for dramatic gains in mass and strength (regardless of quality) may stack Omnadren with heavy orals such as Anadrol or Dianabol. Here of course the strength and weight gain should be even more extreme, although androgenic/estrogenic side effects are expected to be as well.

Although Omnadren stays active in the body for about two weeks, it is generally injected on a weekly or bi-weekly basis. A dosage of 250-750mg per week is more than sufficient to achieve great results. Some take advantage of the very low price of Omnadren (Europe) and take excessively large amounts. Beyond 750mg or 1000mg weekly added side effects will no doubt be greatly outweighing growth, so there is usually little need for such excess. With this drug we really don't want to mistake water bloat for muscle growth. And while a number of adventurous women do experiment with testosterone products, Omnadren is probably not a good choice. The long action of this compound, mixed with the highly androgenic nature of testosterone, makes a poor combination. Virilization symptoms can develop quite easily with a strong androgen, making a long acting product like Omnadren notably dangerous should problems become evident. Testosterone propionate is a much better choice should an androgen like this be absolutely necessary, as it will give the user much greater control over her blood testosterone level.

Due to the extremely low price for this drug in Poland, Omnadren is made readily available on the black market. Among bodybuilders, Omnadren is generally considered to be inferior to Sustanon however. Price may have something to do with this belief, as this drug is usually much cheaper than Sustanon. Counterteits are not a major concern, so it is considered a trustworthy item. Don't be alarmed if your Omnadren seems to have a different look than or you remember from previous experience. The packaging of this product seems to change quite often. For starters, the company name changed officially in 1991 from "Polfa" to "Jelfa", although for years after the older version boxes could still be found in circulation. The color of the boxes also varies, and has included pink & white, green & white, gray, and gray & blue. What is constant is that each box contains five ampules, protected in a white paper or plastic holder. The old ampules are clear glass, and are imprinted with red or black ink that will rub off easily. The newer version is identified by a paper ampule label, which currently bears black print.



 

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rHGH Guide

rHGH Guide
According to studies in the New England Journal of Medicine GH use will:
Shed Bodyfat, Increase Muscle Tone; Boost your Energy, Strength, and Endurance
Reduce Wrinkles and Create Tighter, Smoother Skin; Help you Sleep Better, Improve Sex Drive and Performance, Improve Immune and Heart Function, Bone Density, Healing Time and Cholesterol, Improve Brain Function, Memory and Mental Focus

Wow! Sure sounds like a wonder drug to me! Yeah right, anyway here is some real world information for bodybuilders. Somatropin (rHGH) is produced by the pituitary gland and is responsible in adolescence for growth of tissues, protein deposition, and the breakdown of sub-q fat stores. As we age, growth hormone levels decrease but still remain active in the body, releasing in cycles during the day. Synthetic growth hormone used exogenously by bodybuilders is a 191 chain sequence of amino acids that replicates the bodies natural production of growth hormone.

Growth hormone has been in use by bodybuilders since the early 1980’s, though at this time, HGH was being extracted from the pituitary glands of cadavers and had enormous side effects, most prominently Creutzfeldt Jacob disease. This is a rare and fatal brain disease, it need not be discussed here since it is not possible in synthetic forms of rHGH, but if you want more info just run a search in google. rHGH stimulates growth in most body tissues which is due to an increase in cell number rather than cell size. This includes muscle tissue as well as internal organs, hence the dreaded GH gut.

Use of growth hormone by bodybuilders will cause increased muscle size, localized and overall bodyfat loss, increased protein synthesis, increased glucose output by the liver, increased insulin resistance and lowered thyroid output. Stored fats will be used as a primary fuel source, thus the body fat loss.

So is rHGH the wonder drug everyone lusts after? It certainly is beneficial but not for everyone. You must be willing to take risks to achieve maximum benefits from its use, as well as substantial financial investment. Do it right the first time or don’t do it at all! You will achieve faster and greater growth from cycles of steroids than with GH, though once you reach a plateau, not many products work better.

Ok, so now you have decided that this is the drug for you and you are ready to try it, so what next? Well here are some general guidelines to follow for maximal results from GH use:

Daily injections are a must to maintain stable blood levels as GH has a very short life span in the body. It will peak almost immediately after injection and will clear the body with a half-life of only 20-30 minutes. It is best injected first thing in the morning upon rising to raise levels that are very low from sleeping, and immediately after training. I do not recommend injecting before bed as many bodybuilders do, since that is the time of day that your body will release naturally high levels of growth hormone, and exogenous use will only block that release. If you take it in the morning when levels are low, after training when levels are depleted and then let your body release while sleeping, you are getting one extra release for free! GH is best taken long term, short cycles do not maximize the benefits of muscle cell increase, only fat loss. Here is how I take my GH for maximum benefits:

6iu ed injected sub-q, preferably in the stomach
3iu injected upon rising, 3iu injected immediately post-workout
10iu insulin taken 30 minutes after GH injection
25mcg cytomel ed
use of androgens such as testosterone

The timing of GH and insulin injections is critical. If insulin is injected before the GH, your pancreas will stop release of insulin monitoring due to the exogenous source. GH when injected will mobilize stored glycogen release which will turn into glucose for energy. This will cause a rapid rise in blood sugar levels that will not shut down or stop rising due to the feedback loop being momentarily cut off. You will go hyperglycemic and end up in the hospital. You must first inject your GH, then the insulin; this will cause a rise in glucose release by the GH and will be controlled and shuttled into muscle tissue for repair by the later injection of insulin.
Use of cytomel or some type of T3 hormone is critical since GH use will severely lower thyroid levels. Small exogenous sources are necessary to maintain normal levels and 25 mcg ed is sufficient. This will also aid in body fat loss by maintaining proper thyroid functioning.
Use of androgens is also necessary due to the promotion of anabolism by increasing muscle size that benefits the new cell number increase by the GH. Remember GH will not directly cause muscle cell size increase, just the number of cells, therefore, androgens are necessary to increase size. Testosterone or trenbolone are both highly androgenic and perfect for out stack.

One myth that needs to be cleared up: high doses of GH use and the 5 on 2 off program. First, if you find that you are not achieving results off of 4-6iu ed, than something else is the problem, not your dose. The use of high doses if primarily cause by heat damage to the protein chain causing denatured proteins. This will decrease the effect and you must use higher doses to achieve the same effect. Other reasons for high dose use are; fake gh, not using insulin, cytomel or test, poor diet, improper timing schedule and the 5 on 2 off. This program was recommended by dealers as a way to move product by offering a lower cost cycle. No doctor in the world would recommend this protocol, Peak blood concentrations are reached in 2-6 hours after injection, and therefore, multiple daily injections are necessary to achieve stable release schedule and results. If you take your last injection Friday afternoon, and then not again till Monday morning, then you have negated all effects offered by the 6 hour concentratio. Yes, you will achieve results using a 5 on 2 off program, but not as well as if you inject ed. It’s your money; I can only tell you how to optimize use.

Side effects of GH use include; carpal tunnel syndrome, tingling in the extremities, numbness in the hands and feet, increased organ growth, decreased insulin reception, acromegaly but only in extreme dose use, and decreased thyroid output causing fat accumulation. If you find that you are experiencing any of the above side effects, lower your dose immediately. This is especially important with carpal tunnel. If you feel like your wrists are hurting then lower the dose until pain subsides. You do not want to have that surgery, trust me.

GH is a fantastic product, beneficial for many reasons. Most people will experience thinning of the skin, increased vascularity, fat loss, permanent increases in muscle size due to the cell number increase, and overall feelings of wellness. You will probably need less sleep and feel supercharged all day long. I highly recommend GH use, but only when you have the money to do it right. 4-6 month cycles are optimal, year round if you are over age 35. If you have anything of benefit to add to this thread, feel free to contribute or PM me.

This was posted in the "Very Basix Question on T3/ GH" thread by Mr. Vic. All thanks go to him. But I was wondering if you had any input, especially the final conclusion at the very end of the artice: "If you’ve been using GH without T4, you’ve been wasting half your money – and if you’ve been using it with T3, you’ve been wasting your time. Start using T4 with your GH, and you’ll finally be getting the full results from your investment."

Thyroid Hormone + Growth Hormone
(IF you aren’t using T4 with your GH, you’re not doing it right)
By Anthony Roberts with James Daemon, PhD.

 

Quite some time ago, I wrote a book on Anabolics, and since then, I’ve received quite a bit of feedback on it. Some of the information contained in the book is based on the 50-60 profiles I completed for Steroid.com’s main page. As a result, I get feedback on certain portions of the book from people who have read them online.

When someone takes the time to send an e-mail to Steroid.com or AnabolicBooks LLC, they’re screened, and eventually some of them make their way to my e-mail account. AnabolicBooks LLC is publisher- a little known fact is that my book is actually wasn’t edited by me, nor do I own the rights to any of it. When they forward an e-mail to me, I typically consider it very carefully, and reply to the original sender. If amendments or additions are useful for anything I’ve previously written (readers frequently send me recently published studies), I typically reply and thank the person for their help.

This time, something odd happened. I was forwarded an e-mail from AnabolicBooks, and the reader seemed to know what he was talking about, but (I thought) mistaken about interactions between Growth Hormone and Thyroid medication. I took a look at the e-mail, and knew that I could quickly find a study that I had saved previously, to send to the reader, to verify that the claims in my work on GH were sound.

In this particular case- James Daemon, PhD- was the reader, and was correct in his assessment of the interaction between thyroid hormone and Growth Hormone. And, in direct contradiction, so was I. Thyroid medication decreases the anabolic effect of Growth Hormone. And it increases it.
Huh?

There’s some leaps here, because research in some of the necessary areas is sketchy (or not done yet), but if you read the entirety of this article, you’ll learn how to get a significantly more gains from Growth Hormone, for pennies a day, by the addition of a readily available (and cheap) addition to it. And yeah, it’s a drug you can get anywhere on the ‘net, very easily. And no, it’s not a steroid.
In fact, I’ll go so far as to say you’re throwing away a substantial portion of your gains from growth hormone if you are not using this drug with it.

Ok…I’ll explain things a bit further. First, a brief explanation of Thyroid Hormone as well as Growth Hormone may be necessary.

Your thyroid gland secretes two hormones that are going to be of primary importance in understanding Thyroid/ GH interaction. The first is thyroxine (T4) and the second is triiodothyronine (T3). T3 is frequently considered the physiologically active hormone, and consequently the one on which most athletes and bodybuilders focus their energies on. T4, on the other hand, is converted in peripheral tissue into T3 by the enzymes in the deiodinase group, of which there are three types- the three iodothyronine deiodinase either catalyze the initiation (D1, D2) or termination (D3) of thyroid hormone effects. The majority of the body's T3 (about 80%) comes from this conversion via the first two types of deiodinase, while conversion to an inactive state is accomplished by the third type.

It’s important to note that not all of the body’s T4 is converted to T3, however- some remains unconverted. The secretion of T4 is under the control of Thyroid Stimulating Hormone (TSH) which is produced by the pituitary gland. TSH secretion is in turn controlled through release of Thyrotropin Releasing Hormone which is produced in your hypothalamus. So, when T3 levels go up, TSH secretion is suppressed, due to the body’s self regulatory system known as the “negative feedback loop” . This is also the mechanism whereby exogenous thyroid hormone suppresses natural thyroid hormone production. However, it should be noted that thyroid stimulating hormone (like all other hormones) can not work in a vacuum. TSH also requires the presence of Insulin or Insulin-like Growth Factor to stimulate thyroid function (1) When thyroid hormone is present without either insulin or IGF-1, it has no physiological effect (ibid).

Most people think that T3 is just a physiologically active hormone that regulates bodyfat setpoint and has some minor anabolic effects, but in actuality, in some cases of delayed growth in children, T3 is actually too low, while GH levels are normal, and this has a growth limiting effect on several tissues (2) This could be due to T3’s ability to stimulate the proliferation of IGF-1 mRNA in many tissues (which would, of course, be anabolic), or it could be due to the synergistic effect T3 has on GH, specifically on regulation of the growth hormone gene. Although it is largely overlooked in the world of performance enhancement, regulation of the growth hormone response is predominantly determined by positive control of growth hormone gene transcription which is proportional to the concentration of thyroid hormone-receptor complexes, which are influenced by T3 levels. (3)

At this point, just to give you a better understanding of what’s going on, I think it’s prudent to also give a brief explanation of Growth Hormone ( GH) as well.

Your body’s GH is regulated by many internal factors, such as hormones and enzymes. hormones. A change in the level of your body’s GH output begins in the hypothalamus with somatostatin (SS) and growth hormone-releasing hormone (GHRH). Somatostatin exerts its effect at the pituitary to decrease GH output, while GHRH acts at the pituitary to increase GH output. Together these hormones regulate the level of GH you have in your body. In many cases, GH deficiency presents with a low level of T3, and normal T4(
4). This is of course because conversion of T4-T3 is partially dependant on GH (and to some degree GH stimulated IGF-1), and it’s ability to stimulate that conversion process of T4 into T3.

Interestingly, the hypothalamus isn’t the only place where SS is contained; the thyroid gland also contains Somatostatin-producing cells. This is of interest to us, because in the case of the thyroid, it’s been noted that certain hormones which were previously thought only to govern GH secretion can also influence thyroid hormone output as well. SS can directly act to inhibit TSH secretion or it may act on the hypothalamus to inhibit TRH secretion. So when you add GH into your body from an outside source, you are triggering the body into releasing SS, because your body no longer needs to produce its own supply of GH…and unfortunately, the release of SS can also inhibit TSH, and therefore limit the amount of T4 your body produces.
But that’s not the only interaction we see between the thyroid and Growth Hormone.
As we learned in high-school Biology class, the body likes to maintain homeostasis, or “normal” operating conditions. This is the body’s version of the status quo, and it fights like hell to maintain the comfort of the status quo (much like moderators on most steroid discussion boards). What we see with thyroid/ GH interplay is that physiological levels of circulating thyroid hormones are necessary to maintain normal pituitary GH secretion, due to their directly stimulatory actions. However, when serum concentrations of thyroid hormone increase above the normal range we see an increase in hypothalamic somatostatin action, which suppresses pituitary GH secretion and overrides any stimulatory effects that the thyroid hormone may have had on GH. The suppression of GH secretion by thyroid hormones is probably mediated at the hypothalamic level by a decrease in GHRH release(5).
In addition, as IGF-I production is increased in the hypothalamus after T3 administration and T3 may participate in IGF-1 mediated negative feedback of GH by triggering either increased somatostatin tone and/or decreased GHRH production (6). IGF, interestingly, has the ability to mediate some of T3’s effects independent of GH, but not to the same degree GH can (7.) In fact, IGF-I production is increased in the hypothalamus after T3, administration it may plausibly participate in negative feedback by triggering either increased somatostatin tone and/or decreased GHRH production. So we know that GH lowers T4 (more about this in a sec), but an increase in T3 upregulates GH receptors (8) as well as IGF-1 receptors (9,10).

As can be previously stated, and due to the ability of GH to convert inactive T4 into active T3, GH administration in healthy athletes shows us an entirely predicatble increase in mean free T3 (fT3), and a decrease in mean free T4 (fT4) levels.(11)

Interaction between GH, IGF-I, T3, and GC. GH stimulates hepatic IGF-I secretion and local production of growth plate IGF-I, and exerts direct actions in the growth plate. Circulating T3 is derived from the thyroid gland and by enzymatic deiodination of T4 in liver and kidne.. The regulatory 5'-DI and 11ßHSD type 2 enzymes may also be expressed in chondrocytes to control local supplies of intracellular T3 and GC. Receptors for each hormone (GHR, IGF-IR, TR, GR) are expressed in growth plate chondrocytes.

So, with the use of GH, what we see is an increased conversion of T4-T3, and possible inhibition of Thyroid Releasing Hormone by Somatostatin, and therefore even though T3 levels may rise, there is no increase in T4 (logically, we see a decrease). Now, as we’ve seen, GH is HIGHLY synergistic with T3 in the body, and as a mater of fact, if you’ve been paying any attention up until this point, you’ll note that the limiting factor on GH’s ability to exert many of it’s effects, is mediated by the amount of T3 in the body.

As noted before, T3 enhances many effects of GH by several mechanisms, including (but not limited to): increasing IGF-1 levels, IGF-1 mRNA levels, and finally by actually mediating the control of the growth hormone gene transcription process as seen below:

Comparison of the kinetics of L-T3-receptor binding abundance to changes in the rate of transcription of the GH gene.(3)

As you can see, T3 levels are directly correlative to GH gene transcription. The scientists who conducted the study which provided the graph above concluded that the amount of T3 present is a regulatory factor on how much GH gene transcription actually occurs. And gene transcription is what actually gives us the effects from GH. This last fact really seems to shed some light on why we need T3 levels to be supraphysiological if we’re going to be using supraphysiological levels of GH, right? Otherwise, the GH we’re using is going to be limited by the amount of T3 our body produces. However, since we’re taking GH, and it is converting more T4 into T3, T4 levels are lowered substantially, and this is the problem with GH. and may actually be THE limiting factor on GH…if we assume that at least some of GH’s effects are enhanced by thyroid hormone, and specifically T3, then what we are looking at is the GH that has been injected is being limited by a lack of T3. But that doesn’t make sense, because if we use T3 + GH, we get a decrease in the anabolic effect of GH.

This is where Mr. Daemon, who had contacted me via an e-mail to my publisher, about Thyroid + GH interaction, was able to shed some light on things. You see, I knew that it couldn’t just be the actual presence of enough T3 along with the GH that was limiting GH’s anabolic effect, because, simply adding T3 to a GH cycle will reduce the anabolic effect of the GH (12.).

Originally, he had said to me that T3 was synergistic with GH, wheras I said that T3 actually reduced the anabolic effects of GH- now I realize we were both correct. Logically this presents a bit of a problem, which I believe can be solved. This came from reading several studies provided to me by Dr.Daemon. the trend I was seeing was that even when Growth Hormone therapy was used, T3 levels needed to be elevated in order to treat several conditions caused by a lack of natural growth hormone. And even if the patient was on GH, T3 levels still needed to be elevated. And what I noticed was that those levels were elevated successfully by using supplemental T4 but not T3.

Here’s why I think this is:

Additional T3 is not all that’s needed here. What’s needed is the actual conversion process of T4-T3, and the deiodinase presence and activity that it involves. This is because Local 5'-deiodination of l-thyroxine (T4) to active the thyroid hormone 3,3',5-tri-iodothyronine (T3) is catalyzed by the two 5'-deiodinase enzymes (D1 and D2). These enzymes not only “create” T3 out of T4, but actually regulates various T(3)-dependent functions in many tissues including the anterior pituitary and liver. So when there is an excess of T3 in the body, but normal levels of T4, the body’s thyroid axis sends a negative feedback signal., and produces less (D1 and D2) deiodinase, but more of the D3 type, which signals the cessation of the T4-T3 conversion process, and is inhibitory of many of the synergistic effects that T3 has! Remember, Type 3 iodothyronine deiodinase (D3) is the physiologic INACTIVATOR of thyroid hormones and their effects (13) and is well known to have independent interaction with growth factors (which is what GH and IGF-1 are).(14) This is because with adequate T4 and excess T3, (D1 and D2) deiodinase is no longer needed for conversion of T4 into T3, but levels of D3 deiodinase will be elevated. When there is less of the first two types of deidinase, it would seem that the T3 which has been converted to T4 can not exert it’s protein sparing (anabolic effects), as those first two types are responsible for mediation of many of the effects T3 has on the body. This seems to be one of the ways deiodinase contributes to anabolism in the presence of other hormones.

All of this would explain why anecdotally we see bodybuilders who use T3 lose a lot of muscle if they aren’t using anabolics along with it- they’re not utilizing the enzyme that would regulate some of T3’s ability to stimulate protein synthesis, while they are simultaneously signaling the body to produce an inhibitory enzyme (D3). And remember, for decades bodybuilders who were dieting for a contest have been convinced that you lose less muscle with T4 use, but that it’s less effective for losing fat when compared with T3? Well, as we’ve seen, without something ( GH in this case) to aid in the conversion process, it would clearly be less effective! Since the deiodinase enzyme is also located in the liver, and we see decreased hepatic nitrogen clearance with GH + T3, it would seem that the D3 enzyme is exerting it’s inhibitory effects, but in the absence of the effects of the first two deiodinase enzymes, it remains unchecked and therefore not only limits the GH’s nitrogen retention capability.

In other words, if we have enough to GH in our body aid in supraphysiological conversion of T4 into T3, but we already have the too much (exogenous) T3, the GH is not going to be converting any excess T4 into T3 after a certain point- which would be a limiting factor in GH’s anabolic effects, when coupled with the act that we’ve allowed the D3 enzyme to inhibit the T3/ GH synergy that is necessary.

As further evidence, when we look at certain types of cellular growth (the cartilage cell in this case) we see that GH induced rises in IGF-I stimulates proliferation, whereas T3 is responsible for hypertrophic differentiation. So it would seem that in some tissues, IGF-1 stimulates the synthesis of new cells, while T3 makes them larger. In this particular case, The fact that T4 and (D1) deiodinase is am
active component in this system is noted by the authors. They clearly state (paraphrasing) that: “T4 is is converted to T3 by deiodinase (5'-DI type 1) in peripheral tissues…[furthermore] GH stimulates
conversion of T4 to T3 , suggesting that some effects of GH
may involve this pathway.” The thing I want you to notice is that the authors of this paper state that the that the conversion PATHWAY is probably involved, and not the simple presence of T3. (15 )

Also, that same study notes that T3 has the ability to stimulates IGF-I and expression in tissues that whereas GH has no such effect (ibid).

So what are we doing when we add T3 to GH? We’re effectively shutting down the conversion pathway that is responsible for some of GH’s effects! And what would we be doing if we added in T4 instead of T3? You got it- we’d be enhancing the pathway by allowing the GH we’re using to have more T4 to convert to T3, thus giving us more of an effect from the GH we’re taking. Adding T4 into our GH cycles will actually allow more of the GH to be used effectively!

Remember, the thing that catalyzes the conversion process is the deiodinase enzyme. This is also why using low amounts of T3 would seem (again, anecdotally in bodybuilders) to be able to slightly increase protein synthesis and have an anabolic effect – they aren’t using enough to tell the body to stop or slow down production of the deiodinase enzyme, and hence .Although this analogy isn’t perfect, think of GH as a supercharger you have attached to your car…if you don’t provide enough fuel for it to burn at it’s increased output level, you aren’t going to derive the full effects. Thyroid status also may influence IGF-I expression in tissues other than the liver.So what we have here is a problem. When we take GH, it lowers T3 levels…but we need T3 to keep our GH receptor levels optimally upregulated. In addition, it’s suspected that many of GH’s anabolic effects are engendered as a result of production of IGF-1, so keeping our IGF receptors upregulated by maintaining adequate levels of T3 seems prudent. But as we’ve just seen, supplementing T3 with our GH will abolish Growth Hormone’s functional hepatic nitrogen clearance, possibly through the effect of reducing the bioavailability of insulin-like growth
factor-I (12.)

So we want elevated T3 levels when we take GH, or we won’t be getting ANYWHERE NEAR the full anabolic effect of our injectable GH without enough T3. And now we know that not only do we need the additional T3, but we actually want the CONVERSION process of T4 into T3 to take place, because it’s the presence of those mediator enzymes that will allow the T3 to be synergistic with GH, instead of being inhibitory as is seen when T3 is simply added to a GH cycle. And remember, we don’t only want T3 levels high, but we want types 1 and 2 deiodinase to get us there- and when we take supplemental T3, that just doesn’t happen…all that happens is the type 3 deiodinase enzyme shows up and negates the beneficial effects of the T3 when we combine it with GH.

And that’s where myself and Dr. Daemon ended up, after a week of e-mails, researching studies, and gathering clues.

If you’ve been using GH without T4, you’ve been wasting half your money – and if you’ve been using it with T3, you’ve been wasting your time. Start using T4 with your GH, and you’ll finally be getting the full results from your investment.

Drug Profile: Methandienone

Active Life: 6-8 hours
Drug Class: Anabolic/Androgenic Steroid (Oral)
Average Dose: Men 15-50 mg/day......Women 5-10 mg/day
Acne: Yes, especially in higher dosages
Water Retention: Yes, similar to testosterone
High Blood Pressure: Yes
Liver Toxic: Yes
Aromatization: Yes
DHT Conversion: No
Decrease HPTA function: Yes, dose and cycle length dependant

Methandienone is a derivative of testosterone, exhibiting strong anabolic and moderate androgenic properties. This compound was first made available in 1960, and it quickly became the most favored and widely used anabolic steroid in all forms of athletics. This is likely due to the fact that it is both easy to use and extremely effective. In the U.S. Dianabol production had meteoric history, exploding for quite some time, then quickly dropping out of sight. Many were nervous in the late 80's when the last of the U.S. generics were removed from pharmacy shelves, the medical community finding no legitimate use for the drug anymore. But the fact that Dianabol has been off the U.S. market for over 10 years now has not cut its popularity. It remains the most commonly used black market oral steroid in the U.S. As long as there are countries manufacturing this steroid, it will probably remain so.

Similar to testosterone and Anadrol 50, Methandienone (other known as Dianabol) is a potent steroid, but also one which brings about noticeable side effects. For starters methandienone is quite estrogenic. Gynecomastia is often a concern during treatment, and may present itself quite early into a cycle (particularly when higher doses are used). At the same time water retention can become a pronounced problem, causing a notable loss of muscle definition as both subcutaneous water and fat build. Sensitive individuals may therefore want to keep the estrogen under control with the addition of an anti-estrogen such as Nolvadex and/or Proviron. The stronger drugs Arimidex, Femara, or Aromasin (antiaromatase) would be a better choice if available.

In addition, androgenic side effects are common with this substance, and may include bouts of oily skin, acne and body/facial hair growth. Aggression may also be increased with a potent steroid such as this, so it would be wise not to let your disposition change for the worse during a cycle. With Dianabol there is also the possibility of aggravating a male pattern baldness condition. Sensitive individuals may therefore wish to avoid this drug and opt for a milder anabolic such as Deca-Durabolin. While Dianabol does convert to a more potent steroid via interaction with the 5-alpha reductase anzyme (the same enzyme responsible for converting testosterone to dihydrotestosterone), it has extremely little affinity to do so in the human body's. The androgenic metabolite 5alpha dihydromethandrostenolone is therefore produced only in trace amounts at best. Therefore the use of Proscar/Propecia would serve no real purpose.

Being moderately androgenic, Methandienone is really only a popular steroid with men. When used by women, strong virilization symptoms are of course a possible result. Some do however experiment with it, and find low doses (5mg) of this steroid extremely powerful for new muscle growth. Whenever taken, Methandienone (dianabol) will produce exceptional mass and strength gains. It's effectiveness is often compared to other strong steroids like testosterone and Anadrol 50, and it is likewise a popular choice for bulking purposes. A daily dosage of 20-40mg is enough to give almost anybody dramatic results. Some do venture much higher in dosage, but this practice usually leads to a more profound incidence of side effects. It additionally combines well with a number of other steroids. It is noted to mix particularly well with the mild anabolic Deca-Durabolin. Together one can expect an exceptional muscle and strength gains, with side effects not much worse than one would expect from Dianabol alone. For all out mass, a long acting testosterone ester like enanthate can be used. With the similarly high estrogenic/androgenic properties of this androgen, side effects may be extreme with such a combination however. Gains would be great as well, which usually makes such an endeavor worthwhile to the user. As discussed earlier, ancillary drugs can be added to reduce the side effects associated with this kind of cycle.

In order to withstand oral administration, this compound is c17 alpha alkylated. We know that this alteration protects the drug from being deactivation by the liver (allowing nearly all of the drug entry into the bloodstream), however it can also be toxic to this organ. Prolonged exposure to c17 alpha alkylated substances can result in actual damage, possibly even the development of certain kinds of cancer. To be safe one might want to visit the doctor a couple of times during each cycle to keep an eye on their liver enzyme values. Cycles should also be kept short, usually less than 8 weeks long to avoid doing any noticeable damage. Jaundice (bile duct obstruction) is usually the first visible sign of liver trouble, and should be looked out for. This condition produces an unusual yellowing of the skin, as the body has trouble processing bilirubin. In addition to the skin, the whites of the eyes may also yellow, a clear indicator of trouble. Should this occur the drug should be discontinued immediately and a doctor visited. This is usually a point where further, permanent damage can be avoided.

It is also interesting to note that methandienone is structurally identical to boldenone (EQ), except that it contains the added c17 alpha alkyl group discussed above. This fact makes clear the impact of altering a steroid in such a way, as these two compounds appear to act very differently in the body. The main dissimilarity seems to lie in the tendency for estrogenic side effects, which seems to be much more pronounced with Dianabol. Equipoise is known to be quite mild in this way, and users therefore commonly take this drug without any need of an anti-estrogen. Dianabol is much more estrogenic not because it is more easily aromatized, as in fact the 17 alpha methyl group and c1-2 double bond both slow the process of aromatization. The problem is that methanmdienone converts to l7alpha methylestradiol, a more biologically active form of estrogen than regular estradiol. But Dianabol also appears to be much more potent in terms of muscle mass compared to boldenone, supporting the notion that estrogen does play an important role in anabolism. In fact boldenone and methandienone differ so much in their potencies as anabolics that the two are rarely though of as related. As a result, the use of Dianabol is typically restricted to bulking phases of training while Equipoise is considered an excellent cutting or lean-mass building steroid.

The half-life of PureDbol is only about 3 to 4 hours, a relatively short time. This means that a single daily dosage schedule will produce a varying blood level, with ups and downs throughout the day. The user likewise has a choice, to either split up the tablets during the day or to take them all at one time. The usual recommendation has been to divide them and try to regulate the concentration in your blood. This however, will produce a lower peak blood level than if the tablets were taken all at once, so there may be a trade off with this option. The steroid researcher Bill Roberts also points out that a single-episode dosing schedule should have a less dramatic impact on the hypothalamic-pituitary-testicular axis, as there is a sufficient period each day where steroid hormone levels are not extremely exaggerated. I tend to doubt hormonal stability can be maintained during such a cycle however, but do notice that anecdotal evidence often still supports single daily doses to be better for overall results. Perhaps this is the better option. Since we know the blood concentration will peak about 1.5 to 3 hours after administration, we may further wonder the best time to take our tablets. It seems logical that taking the pills earlier in the day, preferably some time before training, would be optimal. This would allow a considerable number of daytime hours for an androgen rich metabolism to heighten the uptake of nutrients, especially the critical hours following training.